« Previous
Epilepsy Research
Volume 31, Issue 2
, Pages 153-157
, July 1998
Inhibition of [3H]α-amino-3-hydroxy-5-methyl-4-isoxazole-propionic acid [AMPA] binding by the anticonvulsant valproate in clinically relevant concentrations: an autoradiographic investigation in human hippocampus
References
- . Relation of serum valproate concentration to response in mania. Am. J. Psychiatry. 1996;153:765–770
-
Clarenbach, P., Fröscher, W., 1992. Antiepileptika, Neuropsychopharmaka. In: P. Riederer, G. Laux, W. Pöldinger (Eds.), Neurpsychopharmaka, Band 6. Springer Verlag, Wien, pp. 57–84.
- . Excitatory amino acid receptors, neuronal membrane phospholipid metabolism and neurological disorders. Brain Res. Rev. 1991;16:171–191
- . Effects of pharmacological manipulation of GABAergic neurotransmission in a new mutant model of paroxysmal dystonia. Eur. J. Pharmacol. 1991;192:207–219
- . Increased AMPA-sensitive quisqualate receptor binding and reduced NMDA-receptor binding in epileptic human hippocampus. J. Neurosci. 1991;11:428–434
- . Valproic acid selectively reduces the low-threshold (T) calcium in rat nodose neurons. Neurosci. Lett. 1990;116:233–238
- . Excitotoxin l-β-oxalyl-amino-alanine (l-BOAA) and 3,4,6-trihydroxyphenylalanine (6-OH-DOPA) inhibit [3H]alpha-amino-3-hydroxy-5-methyl-4-isoxazole-proprionic acid (AMPA) binding in human hippocampus. Neurosci. Lett. 1994;169:219–222
- . Differential effects of gamma-vinyl GABA and valproate on GABA-transaminase from cultured neurons and astrocytes. Neuropharmacology. 1986;25:617–625
- . Effects of the antiepileptic drug valproate on metabolism and function of inhibitory and excitatory amino acids in the brain. Neurochem. Res. 1993;18:485–502
-
.
Antiepileptic drug mechanism of action.
Epilepsia. 1995;2(Suppl.):2–12
- . Comparison of the structural characteristics of the 4-aminobutyrate: 2-oxoglutarate transaminases from rat and human brain, and of their affinities for certain inhibitors. Biochim. Biophys. Acta. 1978;522:385–399
-
Meldrum, B.S., Garthwaite, J., 1991. Excitatory amino acid neurotoxicity and neurodegenerative disease. In: Lodge, D., Collingridge, G.L. (Eds.), The Pharmacology of Excitatory Amino Acids. A TIPS Special Report, pp. 54–61.
-
.
The effects of AMPA receptor antagonists on kindled seizures and a reflex epilepsy in rodents and primates.
Epilepsy Res. 1992;9(Suppl.):307–311
- . Mechanism of action of antiepileptic and antimyoclonic drugs. Adv. Neurol. 1995;67:329–360
- . The AMPA receptor antagonist NBQX excerts antidystonic effects in an animal model of idiopathic dystonia. Eur. J. Pharmacol. 1993;231:287–291
- . Modulation of the seizure threshold for excitatory amino acids in mice by antiepileptic drugs and chemoconvulsants. J. Pharmacol. Exp. Ther. 1993;265:1063–1070
- . Differential effects of antiepileptic drugs and β-carbolines on seizures induced by excitatory amino acids. Neuroscience. 1990;39:799–807
-
Wilder, B.J., Rangel, R.J., 1989. Clinically relevant antiepileptic drug interactions. In: W.H. Pitlick (Ed.), Antiepileptic Drug Interactions. Raven Press, New York, pp. 65–75.
- . Valproic acid differs in its in vitro effect on glutamic acid decarboxylase in neonatal and adult rat brain. Gen. Pharmacol. 1996;27:635–638
- . Non-NMDA glutamate receptors are involved in the maintenance of status epilepticus. Neuroreport. 1993;5:81–83
PII: S0920-1211(98)00022-9
© 1998 Elsevier Science B.V. All rights reserved.
« Previous
Epilepsy Research
Volume 31, Issue 2
, Pages 153-157
, July 1998
