« Previous
Next »
Epilepsy Research
Volume 29, Issue 3
, Pages 195-200
, February 1998
Vigabatrin enhances promoted release of GABA in neonatal rat optic nerve
References
- . Characterization of [3H]GABA release from striatal slices: Evidence for a calcium-independent process via the GABA uptake system. Neuroscience. 1988;27:563–570
- . Gabapentin potentiates the conductance increase induced by nipecotic acid in CA1 pyramidal neurons in vitro. Epilepsy Res. 1995;20:193–202
- . The anticonvulsant gabapentin enhances promoted release of GABA in hippocampus: A field potential analysis. Brain Res. 1995;692:273–277
-
.
Gamma-vinyl GABA (4-amino-hex-5-enoic acid), a new selective irreversible inhibitor of GABA-T: Effects on brain GABA metabolism in mice.
J. Neurochem. 1977;29:787–802
-
.
Gabapentin increases GABA-induced depolarization in rat neonatal optic nerve.
Neurosci. Letters. 1994;169:181–184
- . GABA levels in the brain: A target for new antiepileptic drugs. Neuroscientist. 1996;6:326–334
- . Bidirectional movement of gamma-aminobutyric acid in rat spinal cord slices. J. Neurochem. 1980;35:1394–1399
- . Transient presence of GABA in astrocytes of the developing optic nerve. Glia. 1993;9:188–198
- . Initial observations of effect of vigabatrin on in vivo 1H spectroscopic measurements of γ-aminobutyric acid, glutamate, and glutamine in human brain. Epilepsia. 1995;36:457–464
- . Two distinct mechanisms, differentially affected by excitatory amino acids, trigger GABA release from fetal mouse striatal neurons in primary culture. J. Neurosci. 1988;9:648–656
- . Nuclear magnetic resonance detection of increased cortical GABA in vigabatrin-treated rats in vivo. Epilepsia. 1994;35:431–436
- 3-Alkyl GABA and 3-alkylglutamic acid analogues: Two new classes of anticonvulsant agents. Epilepsy Res. 1992;11:103–110
PII: S0920-1211(97)00086-7
© 1998 Elsevier Science B.V. All rights reserved.
« Previous
Next »
Epilepsy Research
Volume 29, Issue 3
, Pages 195-200
, February 1998
